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New research gives clinicians better tools to identify preterm survivors at risk of declining lung health

New research from the Wal-yan Respiratory Research Centre points to a more accurate set of warning signs that can flag declining lung function years before it becomes serious.

A mission to make home safer for kids with cystic fibrosis

Dr Kak-Ming Ling, a Wal-yan Respiratory researcher at the Kids Research Institute Australia, has been awarded a $25,000 Illuminate Award for her work to change that. Supported by Conquer Cystic Fibrosis, the award was presented at the 2026 Illuminate PitchFest hosted by The Kids.

Our team

Meet the team at Phage WA, who are working to tackle antimicrobial resistance (AMR) through phage therapy. 

Phage WA Artificial Intelligence Team

Our team uses AI to quickly analyse large amounts of genetic data to help discover alternate medications and improve existing treatments.

Community Involvement

As part of our research development and planning we invite members of the community to work with us. Click here to find out how.

Contact Information

If you have any questions or would like more information about the Western Australian Epithelial Research Program (WAERP), please click here to access our contact details.

Ancestral SARS-CoV-2, but not Omicron, replicates less efficiently in primary pediatric nasal epithelial cells

Children typically experience more mild symptoms of Coronavirus Disease 2019 (COVID-19) when compared to adults. There is a strong body of evidence that children are also less susceptible to Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) infection with the ancestral viral isolate.

Macrophage PD-1 associates with neutrophilia and reduced bacterial killing in early cystic fibrosis airway disease

Macrophages are the major resident immune cells in human airways coordinating responses to infection and injury. In cystic fibrosis, neutrophils are recruited to the airways shortly after birth, and actively exocytose damaging enzymes prior to chronic infection, suggesting a potential defect in macrophage immunomodulatory function.