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This sub-project aims to fill the gap in knowledge of the effects of climate change on child health and wellbeing in Australia, and to propose data-driven adaptation trials to counter the most serious impacts.

Strengths-based, tiered, accessible, resources and supports (STARS) for Kids for parent, carers and their children.

ORIGINS sub-projects are investigating the best way to provide support to new parents and their influence on the health and development of their offspring.

Research shows at least a third of newborns receive formula in hospital, a practice shown to be associated with an increased risk of cow’s milk allergy.

Nutrition in preschool children with autistic behaviours.

Research

Suicide was the leading cause of death among young Australians aged 15-24 years old in 2023, with 392 lives lost. The continued high numbers of youth suicide demand urgent exploration of alternative approaches to suicide intervention in this population. The United Kingdom-based suicide service Maytree offers an innovative short-term stay for people experiencing suicidal thoughts. Grounded by the Maytree model-of-care, the aim of the current study was to co-design a short-stay service responsive to the specific needs of suicidal young people.

Research

Platelets are anucleate blood cells that contain mitochondria and regulate blood clotting in response to injury. Mitochondria contain their own gene expression machinery that relies on nuclear-encoded factors for the biogenesis of the oxidative phosphorylation system to produce energy required for thrombosis.

Research

We aimed to investigate the molecular basis underlying a novel phenotype including hypopituitarism associated with primary ovarian insufficiency.

Research

Aleksandra Filipovska BSc PhD Louis Landau Chair in Child Health Research; NHMRC Leadership Fellow; Deputy Director, ARC Centre of Excellence for

Research

Mutations in the TANGO2 gene cause an autosomal recessive disorder characterised by developmental delay, stress-induced episodic rhabdomyolysis, and cardiac arrhythmias along with severe metabolic crises. Although TANGO2 mutations result in a well characterised disease pathology, the function of TANGO2 is still unknown.